Principle of Wo
an
Flap S
Tse
AADO. H
Nursing sy
ound coverage
nd
Surgery
WL
HKSSH
ymposium
Why Wounds nee
To av
• Infection : Acute or C
• Exposure of importan
vessels, tendon, bon
• Electrolyte loss
• Protein loss
ed to be covered ?
void :
Chronic
nt tissues : nerve,
ne, joint
Consequence
cove
• Uncontrolled sepsis –
• Extensive scarring –
functional deficit
• Ideally, wound cove
achieved within 1 w
of poor wound
erage
– life threatening
cosmetic and
erage should be
week after injury
Management of S
Primary Goal
(initial healing and
infection control)
• Skeletal stability
• Adequate blood
supply
• Soft tissue coverage
Severe limb injury
Secondary goal
(functional
reconstruction)
• Nerve repair/graft
• Muscle & tendon
repair/transfer
• Bone & Joint
reconstruction
Adverse Factors f
• Infection
• Retained foreign bodies
• Retained devitalized tiss
• Poor circulation
• Radiation or chemother
• Systemic factors : malnu
immunosuppression, m
PVD, smoking, obesity
• Wrong choice of treatm
for wound healing
s
sue
rapy
utrition,
medical diseases e.g DM,
ment
Patient pr
• Local :
– adequate debridement
– Dressing care : keep wou
barrier to infection
• Systemic :
– Nutritional support
– Antibiotics
– Pain control
– Stress relief
reparation
und clean and tissue viability,
Dress
• Debridement ( of non viable
tissue)
– e.g. Iroxol
• Decontamination ( of infective
agents)
– Seasorb silver (contain alginate
to trap water)
– Anticoat (silver only)
– Aquacel (hydrofiber)
• Promotion of healing
– Solcoseryl
– Actovergin
– Collagen
• External stimulation :
– Hyphecan
ssing
• Optimal Environment
• Local tissue circulation
Bacter
• All wounds are colonize
• Presence of bacteria ≠
• Established infection do
coverage surgery
– Except certain bacteria
– Streptococcus
• Streptolysin
• Clear thin exudate
– Pseudomonas
• Green colour exudate
riology
ed by bacteria
≠ infection
oes not preclude wound
Bacter
• Rational use of antib
• Nature of wounds
• Intelligent guess
• Avoid prolonged topi
• Surgical debridemen
tissues
riology
biotics
ical antibiotics
nt of dead and infected
Anat
• What structures are m
– Loss of major peripher
• What needs to be rep
– Cavity filling
• What tissues available
– Vascular anatomy
• What tissues available
– Free tissue transfer
• Overall vascular statu
tomy
missing
ral nerves
placed
e nearby
e distant
us of the limb
WOUND PRE
EPARATION
Topical Negative
e Pressure (VAC)
Vacuum Assi
isted Closure
VAC re
• Negative pressure
– Most wound : 125mmHg
– 50-125mmHg for skin gra
• Cycle :
– Constant for 48 hr then in
• Dressing changes
– Most wound : 48 hr then
– Infected : less than 48 hr
– Clean wound : 4-5 days
egime
g
afts
ntermittent (5minon/2min off)
every 4-5 days
r
Reconstruc
• Primary closure
• Secondary intention “wo
• Skin graft
• Flap
– Local
– Distant
– Free
• Select the simple
fulfill wound requ
ction ladder
ound contraction”
est method that
uirement
Skin
• Full thickness (includ
(~0.015 inches)
• Survive on vasculariz
“imbibrition”
• May achieve sensat
sensory nerve into th
graft
ding dermis) or splitted
zed bed by
tion by ingrowth of
he graft
SSG pre
eparation
Tie over
Dona
• Keep dressing intact x
2-3 weeks unless
infection is suspected
ar site
• Failed by poorly vasc
infection, shearing
• Bulky dressing, “Tie o
plaster immobilization
• For SSG : inspect aft
• Skin contracture, hyp
cularized bed,
over dressing”, VAC,
n
ter D5, FTSG : D7
perpigmentation
SS
SG
SSG vs
• SSG
– Depends on the vasculari
– Scars usually bad
– Good for large areas
– Donor sites can be used r
• FTSG
– Limited supply
– Good skin like quality
– Different mechanism of re
– Can be used on bare tend
s FTSG
ity of recipient bed
repeatedly
ecipient site incorporation
don or bone
FLA
AP